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    Prednisone is used for many different autoimmune diseases and inflammatory conditions, including: asthma, COPD, CIDP, rheumatic disorders, allergic disorders, ulcerative colitis and Crohn's disease, adrenocortical insufficiency, hypercalcemia due to cancer, thyroiditis, laryngitis, severe tuberculosis, urticaria (hives), lipid pneumonitis, pericarditis, multiple sclerosis, nephrotic syndrome, sarcoidosis, to relieve the effects of shingles, lupus, myasthenia gravis, poison oak exposure, Ménière's disease, autoimmune hepatitis, giant-cell arteritis, the Herxheimer reaction that is common during the treatment of syphilis, Duchenne muscular dystrophy, uveitis, and as part of a drug regimen to prevent rejection after organ transplant. It is important in the treatment of acute lymphoblastic leukemia, non-Hodgkin lymphomas, Hodgkin's lymphoma, multiple myeloma, and other hormone-sensitive tumors, in combination with other anticancer drugs. Prednisone can be used in the treatment of decompensated heart failure to increase renal responsiveness to diuretics, especially in heart failure patients with refractory diuretic resistance with large dose of loop diuretics. In terms of the mechanism of action for this purpose: prednisone, a glucocorticoid, can improve renal responsiveness to atrial natriuretic peptide by increasing the density of natriuretic peptide receptor type A in the renal inner medullary collecting duct, inducing a potent diuresis. Short-term side effects, as with all glucocorticoids, include high blood glucose levels (especially in patients with diabetes mellitus or on other medications that increase blood glucose, such as tacrolimus) and mineralocorticoid effects such as fluid retention. The mineralocorticoid effects of prednisone are minor, which is why it is not used in the management of adrenal insufficiency, unless a more potent mineralocorticoid is administered concomitantly. It can also cause depression or depressive symptoms and anxiety in some individuals. Deltasone Prednisone Side Effects, Interactions, Warning. Corticosteroid Oral Route, Parenteral Route Proper Use - Mayo Clinic Depo-Medrol Single-Dose methylprednisolone acetate dose.
     
  5. metaContent Well-Known Member

    GH REPLACEMENT therapy has been offered to GH-deficient children for more than 30 yr, but it only became a licensed indication for GH-deficient adults in the United States, a number of European countries, and New Zealand in 1996. Thus, in contrast to the longstanding pediatric literature and interest in the biochemical diagnosis of GH deficiency (GHD), the concerns of the endocrinologist treating adults have been addressed only recently. The type of underlying pathophysiology differs in childhood-onset compared with adult-onset GHD. In childhood, the commonest etiology is isolated idiopathic GHD (1, 2), a blanket term, which includes some children with distinctive pathophysiology that may be demonstrated radiologically, and others in whom the pathological insult is unknown and the explanation for the GHD ill-understood. In contrast, adult-onset GHD is most frequently due to a pituitary adenoma and/or treatment with surgery or radiotherapy (3, 4); isolated idiopathic GHD acquired in adult life has never been reported. In childhood the differential diagnosis is dominated by alternative causes of poor growth (5–9), whereas difficulties in the diagnosis of adult-onset GHD (10–12) exist in the obese and in the elderly. The purpose of this review is to evaluate the usefulness of a variety of clinical and biochemical approaches to the diagnosis of GHD in children and adults. Clonidine-stimulated growth hormone concentrations cut-off values. Growth Hormone Test - Cincinnati Children's Hospital GH Stimulation Tests - labnet.health.nz
     
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